Food For Thought

Worried woman

Where did I leave my keys?

I know I had something important to do today….

I’m sure I know that person, but why can’t I remember their name?

We all have lapses in our memory from time to time. This is a normal part of life that we are all familiar with, especially as we age.  But what happens when it starts to feel like it might be more than a memory lapse?  What happens when our simple forgetfulness seems to be happening more frequently?

Many people fear that their difficulty remembering things could be the first signs of more serious conditions like dementia or even Alzheimer’s disease. While this might be a very real concern for some people, it is very important to remember that these conditions are distinct from the type of forgetfulness that typically comes with aging.

Pensive ManThe fact of the matter is that after a lifetime of meeting new people, learning new things, and
experiencing the world around us – sometimes it just becomes too much for the brain to keep track of.  Think about the brain in the context of a computer hard drive; after years of use, it gets filled with files and starts slowing down.  You have to search longer to find those files that you added when the computer was brand- new.  Sometimes you need to get rid of old files to make room for new information.

Many people would like to avoid deleting these old files – that is to say, they do not want to forget fond memories. What if instead of deleting old files, we could increase the amount of information the hard drive holds? What if we could improve the processor so it can sort through the information faster? The good news, is that the brain is capable of doing just this when we provide it with the nutrients that it needs.

Neuron BrainA renewed focus on the effects of aging on brain health has greatly expanded our knowledge about the biochemical processes behind the processes that can either preserve cognitive function, or lead to a steady decline. We now know that each brain cell contains thousands of mitochondria. These are the “engines” that provide cells with energy to function. Much like an engine, the mitochondria have some very complex mechanisms and when one part breaks, the engine stops working. When mitochondria begin to malfunction, it can lead to premature death of brain cells that are necessary for learning, memory, and many other functions.

Fortunately, we can protect our mitochondria from malfunctioning through directed nutritional support. We are now beginning to understand that certain nutrients and naturally occurring plant-derived compounds can play a significant role in protecting brain cells by protecting the mitochondria from free radical damage.  For the most part, these nutrients can be found naturally in a healthy well-balanced diet.  Unfortunately, there are a number of reasons why most people are not getting adequate amounts of these nutrients in their diet. Additionally, the brain has a natural barrier with the blood that prevents many compounds, including some antioxidants, from entering the brain.

Viniferamine Brain Health was designed specifically to provide the protective nutrients that your brain cells need to resist the types of damage that causes neuro-degenerative diseases.  It utilizes ingredients that are capable of crossing the blood-brain-barrier and exerting their effects on the brain cells where they are needed.

What does the research say about the ingredients used in Viniferamine Brain Health?

Sulforaphane from broccoli extract is a potent antioxidant that has been shown to have positive effects in ischemic (lack of oxygen/blood flow) and traumatic brain injury while also reducing the risk of developing neuro-degenerative diseases.

group-imageECG/EGCG from green tea extract are potent antioxidants that have been shown to improve cognitive function and memory while reducing neuronal damage caused by stress and free-radicals.

Trans-resveratrol from grapevine extract has been shown to reduce the risk of neuro-degenerative diseases and protect neurons from oxidative damage.

Hydroxytyrosol and oleuropein from olive leaf extract have been shown to increase genetic expression of Manganese Superoxide Dismutase (MnSOD) that is one of the cell’s most powerful defenses against free-radical damage.

Fresh turmericCurcumin, extracted from turmeric, is an extremely potent antioxidant with significant evidence supporting its use in many disease states.  It has been shown to protect against neurological damage, and play a role in neurotransmission.

L-taurine is an amino acid with multiple roles in brain health including transmitting nerve impulses (neurotransmission).

Rosavins from Rhodiola Rosea root extract have been shown to reduce the effects of stress in brain cells allowing for improved mental performance in times of stress.

SynapseN-acetyl-L-cysteine (NAC) is an amino acid that is essential for the production of glutathione – one of the cell’s most important antioxidants and detoxifying molecules.  It is vital for the removal of oxidative free-radicals and metabolic wastes that can accumulate and damage brain cells.  Low glutathione levels have been associated with increased risk of neuro-degenerative disorders while supplementation with NAC has been shown to decrease the risk.

B-Vitamins including B6, B12, and folic acid are vital to the growth and development of brain cells and nerves. Deficiencies during pregnancy are known to result in severe birth defects and mental retardation.  Deficiencies over a lifetime have been associated with increased risks of neuro-degenerative disorders.

Magnesium is used in more than 300 biochemical reactions in the body, making it amongst the most important minerals in the body.  Without it, cells cannot divide and nerve impulses cannot be sent – leading to atrophy and cell death.

For even more brain benefits, try adding Viniferamine Detox Support, Mood Support, Sleep Support, and Energy Support

References:

  1. Adair JC, Knoefel JE, Morgan N. Controlled trial of N-acetylcysteine for patients with probable Alzheimer’s disease. Neurology. 2001;57(8):1515–1517.
  2. Ames BN, Liu J. Delaying the mitochondrial decay of aging with acetylcarnitine. Ann N Y Acad Sci. 2004;1033:108–16.
  3. Darbinyan V, Kteyan A, Panossian A. Rhodiola rosea in stress induced fatigue—A double blind cross-over study of a standardized extract SHR-5 with a repeated low-dose regimen on the mental. Phytomedicine. 2000; 7(5):365-71
  4. El Idrissi A. Taurine improves learning and retention in aged mice. Neurosci Lett. 2008;436(1):19–22.
  5. Féart C, Samieri C, Rondeau V, et al. Adherence to a Mediterranean diet, cognitive decline, and risk of dementia. JAMA. 2009;302(6):638–48.
  6. Haque AM, Hashimoto M, Katakura M, Tanabe Y, Hara Y, Shido O. Ingestive Behavior and Neurosciences Long-Term Administration of Green Tea Catechins Improves Spatial Cognition Learning Ability in Rats 1. Ingestive Behav Neurosci. 2006;136:1043–1047.
  7. Lemke MR. Plasma Magnesium Decrease and Altered Calcium / Magnesium Ratio in Severe Dementia of the Alzheimer Type. Biol Psychiatry. 1995;3223(94).
  8. Louzada PR, Paula Lima AC, Mendonca-Silva DL, Noël F, De Mello FG, Ferreira ST. Taurine prevents the neurotoxicity of beta-amyloid and glutamate receptor agonists: activation of GABA receptors and possible implications for Alzheimer’s disease and other neurological disorders. FASEB J. 2004;18(3):511–8.
  9. Martínez M, Hernández a I, Martínez N. N-Acetylcysteine delays age-associated memory impairment in mice: role in synaptic mitochondria. Brain Res. 2000;855(1):100–6.
  10. Mattson MP, Chan SL, Duan W. Modification of brain aging and neurodegenerative disorders by genes, diet, and behavior. Physiol Rev. 2002;82(3):637–72.
  11. Milgram NW, Araujo J a, Hagen TM, Treadwell B V, Ames BN. Acetyl-L-carnitine and alpha-lipoic acid supplementation of aged beagle dogs improves learning in two landmark discrimination tests. FASEB J. 2007;21(13):3756–62.
  12. Müller WE, Eckert A, Kurz C, Eckert GP, Leuner K. Mitochondrial dysfunction: common final pathway in brain aging and Alzheimer’s disease–therapeutic aspects. Mol Neurobiol. 2010;41(2-3):159–71.
  13. Ogle WO, Speisman RB, Ormerod BK. Potential of Treating Age-Related Depression and Cognitive Decline with Nutraceutical Approaches: A Mini-Review. Gerontology. 2012:1–9.
  14. Tarozzi A, Angeloni C, Malaguti M, Morroni F, Hrelia S, Hrelia P. Sulforaphane as a potential protective phytochemical against neurodegenerative diseases. Oxid Med Cell Longev. 2013;2013:415078.
  15. Xu Y, Lin D, Li S, et al. Curcumin reverses impaired cognition and neuronal plasticity induced by chronic stress. Neuropharmacology. 2009;57(4):463–71.

Disclaimer: These statements have not been reviewed by the FDA. Viniferamine products are dietary supplements and are not intended to treat, cure, or prevent any disease. The decision to use these products should be discussed with a trusted healthcare provider. The authors and the publisher of this work have made every effort to use sources believed to be reliable to provide information that is accurate and compatible with the standards generally accepted at the time of publication. The authors and the publisher shall not be liable for any special, consequential, or exemplary damages resulting, in whole or in part, from the readers’ use of, or reliance on, the information contained in this article. The publisher has no responsibility for the persistence or accuracy of URLs for external or third party Internet websites referred to in this publication and does not guarantee that any content on such websites is, or will remain, accurate or appropriate.

Copyright 2014 McCord Research – All Rights Reserved.

Leave a Reply

Fill in your details below or click an icon to log in:

WordPress.com Logo

You are commenting using your WordPress.com account. Log Out / Change )

Twitter picture

You are commenting using your Twitter account. Log Out / Change )

Facebook photo

You are commenting using your Facebook account. Log Out / Change )

Google+ photo

You are commenting using your Google+ account. Log Out / Change )

Connecting to %s